10:54,2,Dec,2009 | (1334/0/0) | Original
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Overview of renal sclerosis nephroscleria, nephrosclerosis, including renal artery atherosclerotic renal artery caused by sclerosis, and hypertension caused by benign nephrosclerosis and malignant nephrosclerosis. Renal artery atherosclerosis is systemic atherosclerosis part of the more common in the elderly, with or without hypertension. Causes 1, arterial nephrosclerosis: Pathogenesis and other organs the same as atherosclerosis. 2, benign nephrosclerosis: hypertensive vascular lesions and glomerular hypertension can lead to glomerular injury. Benign renal vascular and glomerular sclerosis lesions are also seen in the elderly, even in patients without hypertension. These changes may be decreased and the elderly on GFP. 3, malignant nephrosclerosis: the initial lesion is due to the rapid increase in blood pressure caused by the vessel wall injury. Symptoms of renal arterial systemic sclerosis patients can be seen the performance of atherosclerosis, such as heart, brain, and the performance of peripheral vascular sclerosis, with or without hypertension. Benign essential hypertension are overweight on the performance, headache, dizziness, palpitations, shortness of breath, mental stress, and chest pain and so on. The performance of a vicious headache and hypertensive encephalopathy, weight loss, visual disturbances. Early nephrosclerosis see backache, low back pain, edema, hematuria, nocturnal enuresis, etc., see the late performance of renal failure. Renal arterial systemic sclerosis patients see the performance of atherosclerosis, such as heart, brain, and the performance of peripheral vascular sclerosis, with or without hypertension. Benign essential hypertension often of moderately elevated blood pressure, leading to congestive heart failure or cerebral vascular accident, only a small number of patients died of renal failure. Malignancy of the diastolic blood pressure 130mmHg or more often, papilledema its outstanding features, and sometimes bleeding and exudate can be seen, often leads to kidney failure. Check 1, blood, urine. Urinalysis: â‘ proteinuria. â‘¡ red blood cells: mainly seen in patients with malignant phase, severe gross hematuria was 2, the renal function: the benign stage patients, mild to moderate hypertension usually has no effect on renal function, more severe hypertension reduced to 65ml majority of the GFP / sub-level, in the event of renal insufficiency before the earlier of hyperuricemia may be the performance of the artery and small artery disease caused by a reflection of reduced renal blood flow. 3, renal biopsy. Treatment 1, symptomatic treatment. 2, active treatment of the primary disease. Renal sclerosis patients generally good prognosis, only a small number of cases to the development of renal failure.
Renal sclerosis, including atherosclerotic
Nephrosclerosis diet because of the attention:
1, the absolute prohibition.
2, use skim milk or yogurt.
3, egg yolk a day to eat no more than 2.
4, hanged animal oil; the total amount of vegetable oil is not more than 20 grams.
5, do not eat animal organs (ie, water, unloading), chicken skin, fat and roe, Crab.
6, eat fried food.
7, do not eat chocolate.
8, eat less fat in the soy and gluten.
9, the daily consumption of 500 grams of fresh green vegetables.
10, the staple food to eat fruit to reduce food intake after the Japanese eat an apple, 50 grams of staple food should be reduced.
11, yams, sweet potatoes, taro potatoes, etc., with the staple food rice, flour, food exchange, the total should be limited.
12, daily salt intake to 5 - 6 grams limit.
13, onion, garlic, ginger, peppers and other "four hot," to eat, but should not eat.
14, regularly eat fish, shrimp and other seafood.
15, lipid-lowering foods include: oats, millet and other coarse grains, black sesame, black fungus, kelp, broccoli and other green hair Levin and fresh vegetables.
16, should be eating dinner, avoid snacks before going to sleep.
17, 30 grams daily with hawthorn, cassia seed 15 grams of grass, water 1000 ml behalf of the tea.
18, if the fatty liver caused by liver dysfunction, or elevated aminotransferases should be under the guidance of a doctor taking lipid-lowering drugs, fish oil JiangMei drugs and health products, but not too much to take. Kidney stones diet should note that (a ) to drink boiled water and more water to be diluted urine, calcium and oxalate concentration is reduced, can not form calcium oxalate stones. research shows that 50% of the urine, kidney stones can decrease the incidence of 86%.
(B) a reasonable calcium, particularly calcium diet on patients with kidney stones tend to "talk about the mere mention of calcium", mistakenly believe that the culprit is calcium kidney stones, is not the case, patients also need calcium kidney stones. At present the medical profession from two different perspective to explain why patients with calcium kidney stones.
The first is to supplement the calcium in the gastrointestinal tract and vegetables contain oxalic acid together to form insoluble calcium oxalate, with the feces in vitro, was partially reduced gastrointestinal absorption and oxalic acid excreted by the kidneys, thus reducing the chance of forming kidney stones.
The second is the Japanese scholar's "theory of acid-base balance." When the blood acidic, easy to form stones. Was alkaline, the inhibition of stone formation. Calcium in the blood acidic, reasonable calcium, blood slightly alkaline, so instead beneficial to inhibit stone formation.
(C) limited intake of carbohydrates to a recent study U.S. scientists showed that the intake of high-sugar foods may increase the chance of kidney stones, therefore, should pay attention to eating sweets.
(D) eat foods high in oxalate content of foods with high oxalate containing tomato, spinach, strawberries, beets, chocolate, excessive intake of oxalate is one of the main causes of kidney stones.
(E) eating soy products containing soy foods are high oxalate and phosphate, calcium can be fused with the kidney to form stones.
(F) carefully before going to bed sleep bad people drink milk, a glass of milk before bed can help sleep. But in sleep, decreased urine output concentrated urine increased in a variety of physical material. And after 2 or 3 blues hours, it is the peak of calcium excretion by the kidneys. calcium through the kidneys suddenly increased in a short time, easy to form stones. Thus patients with kidney stones, high calcium before bed should not drink milk.
(Vii) Do not overdose cod liver oil cod liver oil is rich in vitamin D, calcium and phosphorus can promote intestinal absorption of membrane function, a sudden increase in urine excretion of calcium and phosphorus is bound to have precipitation, easy to form stones.
(H) eat black fungus black fungus is rich in minerals and trace elements, all kinds of stones can have a strong chemical reaction, so that stones stripping, differentiation, dissolved body.
Overview: What is the renal atherosclerosis?
Kidney is due to renal artery atherosclerosis and branch and / or small arteries should be affected by renal function of a class of diseases. According to the speed of progression of renal arteries were divided into benign and malignant small arterial sclerosis glomerulosclerosis.
Causes: Renal atherosclerosis is what causes?
Benign arteriolar nephrosclerosis is the main cause of hypertension, old age. As a result of long-term hypertension in small renal arteries.
Malignant small renal arteries hardening of underlying diseases as high blood pressure about 40%, 15% of chronic nephritis, the rest is multiple nodules arteritis, radiation nephritis, congenital renal disease, renal pelvis fluid, Cushing syndrome.
Symptoms: What are the clinical manifestations of renal atherosclerosis?
1. Often moderate degrees of hypertension and its clinical symptoms. 2. May be associated with heart, brain and retinal arteriosclerosis and its performance. 3. With mild proteinuria, hypertension was positively correlated with the degree. 4. Late may have nocturia, urine specific gravity and urine infiltration decreased, so that the performance of renal insufficiency.
Check: check renal arteriosclerosis What should be done?
1.50 years of age or long-term high blood pressure in patients not well controlled, moderate degrees of hypertension. 2. Mild proteinuria, with the main renal tubular dysfunction. 3. Associated with cardiac, cerebral arteriosclerosis performance and fundus changes. 4. The exclusion of other renal disease (such as chronic nephritis) caused by high blood pressure.
Treatment: kidney arteriosclerosis should be treated?
1. Early hypertension, and normal oral diuretic-based antihypertensive agents. Select one or two each time, invalid sequence replacement, diuretics, Î²-blockers, calcium antagonists and converting enzyme inhibitors.
2. In the latter part of high blood pressure by the "Duplex" or "triple" and use of antihypertensive drugs, combined with lipid-lowering drugs. When necessary, intravenous vasodilators.
3. If the renal dysfunction, in addition to proper blood pressure, but also refer to the treatment of renal dysfunction.
4. Symptomatic and supportive treatment, and
5. Uremia advanced renal dysfunction, with the dialysis treatment.
Prevention: Prevention of kidney arteriosclerosis How?
Renal arteriosclerosis, secondary to hypertension, Clinical divided into benign and malignant renal artery renal sclerosis, the former longer course, the general course of 20 years, but in the course of illness is still a small number of patients (1% -8% ) into malignant stage, if not promptly treated, often death in 1-2 years, the rapid development of the latter disease, renal function deteriorated sharply in the short term into renal failure with uremia, often accompanied by the determination, brain and other organs function decline, If the failure to death within 3 months of treatment more than 50%, 90% within 1 year.
With early patient treatment, the key is to make long-lasting control of blood pressure within the normal range in most patients the renal artery sclerosis benign prognosis is optimistic.