14:25,18,Mar,2009 | (967/0/0) | Original

parietal lobe tumor symptoms


What are the performance and parietal lobe tumors diagnosed?
Parietal lobe damage caused when a tumor is mainly sensory dysfunction contralateral to lesions, and involvement of the visual and language functions.
1. Sensory dysfunction sensory dysfunction, sensory disturbance, and general sub-cortical sensory disturbances, usually caused by parietal lobe tumor pain, temperature sense obstacles and more obvious, even if there is, also occurred in the distal limbs, was very mild type of gloves or socks The sensory dysfunction, which is also accepted as part of the hypothalamus pain, temperature sensation caused by the impulse, cortical sensory dysfunction as a disease mainly contralateral limb position sense, two point discrimination sense, tactile sense of positioning and map-type obstacles, such as patient In the eyes closed cases, holding the hands of the objects, although they would feel, but can not determine the object's weight, size, shape, texture, and even the skin can not write simple number recognition, therefore, can not completed a comprehensive analysis of the object, called the loss of physical sense, it is back to the top of the central lobule after extensive destruction caused by the results of cortical sensory dysfunction can also be manifested as sensory neglect, when the stimulus affected limb, it feels completely normal or slightly decreased, while stimulating both limbs, such as, only the feelings caused by the contralateral limb, while the affected limb completely ignored, when the tactile stimulation of the factors that affected limb has been removed, the patient is still in a period of time felt by the stimulate the feeling persists, the phenomenon known as tactile retention.
2. Body image disturbance autologous patients have difficulty in understanding the structure, which is especially prevalent right parietal lobe lesions, to elucidate the mechanism has not yet been fully understood, many clinical manifestations, such as patients with paralysis of their own indifference, do not pay attention, as if has nothing to do with, meaning no anxiety, loss of Notes called hemiplegia syndrome, the patient completely deny their hemiplegia, paralysis of the limbs even when the prompts to patients, strongly denied that it was his own body, sometimes that is someone else's hand or foot, or to explain the reasons unrelated to physical activity can cause paralysis, said the phenomenon does not know the disease, some patients had lost limbs in the sense that his body no longer exists, the paralyzed limbs is not their own, their limbs have been lost in this, also characterized by a feeling more of a body or more, this show is called phantom limb syndrome and more, plus patients had finger agnosia, left and right body disorientation, amnesia, etc. from the body.
3. Loss of structural disorder also known as loss of structural disorder apraxia structure means the structure of the arrangement of space objects, architecture, pain
ting, design and other issues involving the relationship between space and can not correctly understand and identify, can not be combined, can not understand each other, the the relationship between the use of tools is not working properly, resulting in life was difficult, the clinical applicability of painting, building blocks and other methods of examination, the patient although the sink, and each component may still, but the ratio between the lack of capacity and layout, or upside down, left, is too crowded or scattered arrangement, loss of original shape, there is no concept of space, the lack of three-dimensional relationship between the loss of structure, mechanism of disease production has not yet achieved consensus.
4.Gerstmann syndrome seen after the lower part of the angular gyrus parietal lobe, supramarginal gyrus in the occipital and parietal parts of transitional lesions, the clinical manifestations finger agnosia, loss of orientation about disease, loss of writing, the main miscalculation , finger agnosia most common, often bilateral, instruct patient to produce the specified fingers, the fingers can not be identified, using the chaos of the fingers, especially thumb, little finger, middle finger the most serious, finger agnosia is the Gerstmann syndrome important part of the disease is not only about loss of orientation in the identification of self when others can not distinguish between left and right limbs, but about the surrounding environment is not necessarily affect the orientation, agraphia, mainly it difficult to write, but reading or copying can not be obstacles, significant obstacles acalculia to written calculation.
5. Alexia top of the left cerebral hemisphere occipital often caused by dyslexia, the reading disability, accompanied by writing disabilities, dyslexia can be divided into two types:
(1) subcortical alexia: the patient can not read written or printed text and can not be pronounced, but the spontaneous writing and copying without barriers, patients can rely on writing to express their thinking, but you can not read his articles written or letters, often accompanied by subcortical alexia have hemianopia.
(2) cortical alexia: Patients do not know and can not be read than text, but also often accompanied by written any errors, and not dictation, transcription and spontaneous writing.
6. Parietal lobe epilepsy seizures, tumors, mostly due to the limitations of the attack, and often feel, performance for the onset of contralateral lesions paresthesia, starting with the thumb and index finger more common parts, but the foot is not started by rare, with paroxysmal numbness, electric shock-like sensation or pain-based, extended to the fixed direction, but it was limited for the sport or clonic spasm, or sensory symptoms before the symptoms start following the onset of exercise, even evolved into grand mal epilepsy, often after the onset point discrimination sensation, physical sensation, position sense and other transient sensory disturbance.
7. Hemiplegia or paralysis parietal lobe tumors often appear single lesions or a single contralateral limb paralysis, hemiplegia, paralysis is not a symptom of parietal lobe itself is invaded and the motor areas of the tumor due to forward, and paralysis symptoms at the same time, we can see deep hyperreflexia, increased muscle tone but not obvious.
8. Other lobe tumor, and sometimes there can be muscle atrophy lesions of the contralateral limb, visual distortion produced by optical illusion, the opposite 1 / 4 blind or with bits of hemianopia, as well as geographical awareness of the barriers between .
parietal lobe tumor symptoms

【Overview】
The incidence of cancer than the amount of parietal and temporal lobes are low, tumors were gliomas, accounting for 8.52% of intracranial gliomas; followed by meningiomas, accounting for 6.50% of intracranial meningiomas; then followed for the transfer tumor. Occurs in adults. Parietal function very complex, it is mainly analyze, synthesize a variety of sensory information in order to identify and determine the nature and location of stimulation. Therefore, the parietal lobe damage to the tumor showed the opposite half-length of the main sensory dysfunction, most patients can be sure of the signs and symptoms occur, especially associated with the limitations of sensory seizures, the diagnosis is more positioning Source: Medical Education Network . After several decades, the loss of the structure of the parietal lobe syndrome, hemiplegia feel ignorant disease, loss of orientation disease, Gerstmann syndrome, study, and people recognize the symptoms of parietal lobe has been further improved. Based on the patient symptoms and signs, can make diagnosis more accurate positioning. However, there a few cases, particularly slow-growing meningioma, even though most of the parietal lobe involvement can also be no obvious symptoms.
Clinical manifestations】 【Collecting Medical Education Network
Parietal lobe damage caused when a tumor is mainly sensory dysfunction contralateral to lesions, and involvement of the visual and language functions. 1. Sensory dysfunction sensory dysfunction, sensory disturbance, and general sub-cortical sensory impairments. Parietal lobe tumors usually caused by pain, temperature sense obstacles and more obvious, even if there is, also occurred in the distal limb, showing a very slight feeling of gloves or socks-type obstacles. It is also accepted as part of the hypothalamus pain, temperature sensation caused by the impulse. Cortical sensory dysfunction as a disease mainly contralateral limb position sense, two point discrimination sense, tactile sense of positioning and obstacle graphics. If a patient is in the eyes closed cases, holding the hands of the objects, although they would feel, but can not determine the object's weight, size, shape, texture, and even the skin can not write simple number recognition, so , can not complete a comprehensive analysis of the object, called the loss of physical sense, it is back to the top of the central lobule after extensive destruction caused by the results. Cortical sensory dysfunction can be manifested as sensory neglect, when the stimulus affected limb, it feels completely normal or slightly decreased, while stimulating both limbs, such as, only the feelings caused by the contralateral limb, while the affected limb completely ignored. Ipsilateral limb when the tactile stimulation factors have been removed, the patient is still in a period of time feel the feeling stimulated by the continued existence of the phenomenon known as tactile retention. 2. Body image disturbance autologous patients have difficulty in understanding the structure, which is especially prevalent right parietal lobe lesions, to elucidate the mechanism has not yet been fully understood. Many clinical manifestations, such as patients with paralysis of their own indifference, do not pay attention, if and has nothing to do, there is no meaning of anxiety, called hemiplegia lost NOTE disease. The patient completely deny their hemiplegia, paralysis of the limbs even when the tips to the patients, strongly denied that it was his own body, sometimes considered to be someone else's hands or feet, or to explain the physical reasons unrelated to the reason is not in such phenomenon does not know the disease known as hemiplegia. Some patients had lost limbs in the sense that his body no longer exists, is not paralyzed his body, his limbs this has been lost. Another performance is more a physical feeling or more, this show is called phantom limb syndrome and more. Another patient had finger agnosia, disorientation around the body, such as autologous amnesia. 3. Loss of structural disorder, also called loss of structure, structural disorder apraxia. Refers to the structure of the arrangement of space objects, architecture, painting, design and other issues involving the relationship between space and can not correctly understand and identify, can not be combined, can not understand the relationship between each other and can not properly use the tools to work, resulting in life find it difficult to . Available clinical painting, building blocks and other methods to check the patient although sink, and each component may still, but the ratio between the lack of capacity and layout, or up and down upside down, overcrowded or decentralized arrangement, loss of original shape, no The concept of space, the lack of solid relationship. Resulting loss of structure, mechanism of disease has not yet achieved consensus. 4.Gerstmann syndrome seen in the lower part of the angular gyrus after parietal lobe, supramarginal gyrus in the occipital and parietal parts of transitional lesions, the clinical manifestations finger agnosia, loss of orientation about disease, loss of writing, the main miscalculation . Finger agnosia most common, often bilateral, instruct patient to produce the specified fingers, the fingers can not be identified, using the chaos of the fingers, especially thumb, little finger, middle finger the most serious, finger agnosia Gerstmann syndrome is important components. About loss of directional disorder not only in the identification of self to others when they can not distinguish between left and right limbs, but about the surrounding environment, orientation is not necessarily affected. Agraphia, mainly it difficult to write, but can not read or copied obstacles. Acalculia barriers to written calculation significantly. 5. Alexia top of the left cerebral hemisphere occipital often caused by dyslexia, the reading disability, accompanied by writing disabilities. Dyslexia can be divided into two types: (1) subcortical alexia: the patient can not read written or printed text and can not be pronounced, but the spontaneous writing and copying from obstacles. Patients can rely on writing to express their thinking, but you can not read their own written articles or letters. Subcortical alexia often accompanied by hemianopia. (2) cortical alexia: Patients do not know and can not be read than text, but also often accompanied by written any errors, and not dictation, transcription and spontaneous writing. 6. Parietal lobe epilepsy seizures, cancer, mostly due to the limitations of the attack, and often feel, performance for the onset of contralateral lesions paresthesia, starting with the thumb and index finger more common parts, but the foot is not started by rare, with paroxysmal numbness, electric shock-like sensation or pain-based, extended to the fixed direction, but it was limited for the exercise of muscle spasm or clonus, or the first symptoms begin to feel symptoms following the onset of exercise, even evolved into grand mal epilepsy. Often point discrimination after the onset of sleep. Physical sensation, position sense and other transient sensory disturbance. 7. Hemiplegia or paralysis parietal lobe tumors often appear single lesions or a single contralateral limb paralysis, hemiplegia. Symptoms of paralysis is not the parietal lobe itself is invaded and the motor areas of the tumor due to forward. And paralysis symptoms the same time, we can see deep hyperreflexia, increased muscle tone but not obvious. 8. Other lobe tumor, and sometimes there can be muscle atrophy lesions of the contralateral limb, visual distortion produced by optical illusion, the opposite 1 / 4 blind or with bits of hemianopia, as well as geographical awareness of the barriers between . 】 【Auxiliary examination
1. Skull plain film parietal lobe tumor and cerebral hemispheres, like other parts of the tumor, according to the different nature of the tumor, some tumors may also cause varying degrees of changes in the skull, such as small meningioma of the skull can cause bone growth or inner plate destruction. 2. Ultrasound more common in parietal lobe tumor with ultrasound waves to the contralateral midline shift. 3. Electroencephalogram EEG parietal lobe tumors performance characteristics are: the limitations of δ or θ wave wave appears broader, generally the top side of the main disease, spread to the occipital, temporal, the temporal ago, the amount of , the top part of the lower, especially at the top of the brain falx tumors, slow the emergence of a wider, often difficult to distinguish tumor occipital and temporal. δ wave confined to the top or pillow top, there are likely to parietal lobe tumor. Deep parietal lobe tumor, usually not obvious limitations of δ wave, multi-display 5 to 7 times / second limitation θ scattered or continuous wave and lazy wave. Parietal lobe tumors easily lead to disease hemisphere (30%) or both sides of the hemisphere (about 10%) of the broad diffuse slow wave, sometimes generalized spike and wave display, and the case or the background on both sides of α wave often disorders. 4. Parietal cerebral angiography Cerebral angiography of tumors is characterized by: the before and after the bit as the anterior cerebral artery was parallel to the side of the shift, the convex branch of the middle cerebral artery or in part, to the down separately. In the artery as the lateral under pressure, where the parts of the tumor blood vessels under pressure. (1) parietal falx brain tumors: before and after the bit like the shift to the opposite side shows artery for the straight line, the remote shift significantly. Lateral side as the tumor before the show, the corpus of peripheral artery and its branches curved; tumor after partial arterial end of the week when the corpus has been under pressure to move or corpus of peripheral arterial pushed forward bending, straightening, or were terminal branches arc shape. Artery group of pushing forward a little down, or peripheral dispersion phenomenon. (2) parietal parasagittal tumor: as shown before and after the bit shift to the contralateral artery, the top was, under the pressure of the corpus of peripheral arterial branches curved, falx sign positive. Middle cerebral artery groups were under pressure to low. Lateral groups like the see the downward shift in the arteries, the corpus of peripheral arterial slightly down or flat, so place mostly meningiomas, Gu Chang said in the anterior cerebral artery and tortuous artery thickening. (3) frontoparietal tumors: before and after the show bit like an arc-shaped anterior cerebral artery was shifted to the opposite side. Middle cerebral artery group shifted slightly down. No changes in the blood vessels "○" shaped performance. Lateral middle cerebral artery, as shown flattened obvious pressure, changes in the amount of lifting arteries was significantly higher than other parts of the tumor, manifested by the extrusion, straightening, branch separated from each other and so on. 5. Ventriculography due to the back and triangular lateral body corresponds with the parietal lobe, parietal lobe tumors show it under the lateral pressure of the body, septum pellucidum, third ventricle was shifted to the opposite side slash. After partial tumor close to the occipital, parietooccipital tumors that can be caused by both Delta and occipital horn down. 6. CT brain CT examination of intracranial tumors, especially in the diagnosis of supratentorial tumors than other diagnosis techniques have more advantages, is the ideal method of diagnosis. 】 【Differential diagnosis
(A) of the chronic subdural hematoma parietal chronic subdural hematoma is a common disease in both young common. The clinical course and brain tumors are similar, but have history of head injury, the more minor injuries, it is often forgotten by the patient and not take the initiative to tell, even when asked not remember history. After more than a few months or even years before clinical symptoms appear gradually. Early symptoms are usually headache, dizziness, epilepsy can also find out the limitations of sensory or sensory disability side of the body, careful examination can find some signs and symptoms of parietal lobe damage. Skull plain film examination may show chronic increased intracranial pressure, usually no fracture line. Ultrasound examination revealed midline wave shift (no shift may be bilateral). EEG slow wave appears to have limitations. Carotid angiography showed no vascular area can often confirm the diagnosis. (B) of the parietal lobe brain abscess, the incidence of brain abscess than temporal, frontal, far more than the blood-borne infection, acute expression of more than a systemic (fever, peripheral blood polymorphonuclear leukocytes as an increase) and acute intracranial pressure by other, lumbar puncture showed purulent CSF has changed, it is easy and differential diagnosis of brain tumors. However, chronic parietal lobe brain abscess, longer course, the similar clinical symptoms and parietal lobe tumors, most difficult to identify, only when surgical exploration to confirm the diagnosis. (C) of the cerebral vascular lesions in the brain artery disease, such as vascular malformations, arteriovenous fistula, aneurysm, arteriosclerosis, etc., there may be parietal lobe syndrome. The top of the middle cerebral artery, temporal, occipital lobe occupies prone to ischemic softening. Dominant hemisphere angular gyrus softening beneath writing, reading and loss of Gerstmann syndrome occurs, care should be carefully examined and tumors.
.
After several decades, the loss of the structure of the parietal lobe syndrome, hemiplegia feel ignorant disease, loss of orientation disease, Gerstmann syndrome, study, and people recognize the symptoms of parietal lobe has been further improved. Based on the patient symptoms and signs, can make diagnosis more accurate positioning. However, there a few cases, particularly slow-growing meningioma, even though most of the parietal lobe involvement can also be no obvious symptoms. Performance; parietal lobe damage caused when a tumor is mainly sensory dysfunction contralateral to lesions, and involvement of the visual and language functions. 1. Sensory dysfunction; sensory dysfunction, sensory disturbance, and general sub-cortical sensory impairments. Parietal lobe tumors usually caused by pain, temperature sense obstacles and more obvious, even if there is, also occurred in the distal limb, showing a very slight feeling of gloves or socks-type obstacles. It is also accepted as part of the hypothalamus pain, temperature sensation caused by the impulse. Cortical sensory dysfunction as a disease mainly contralateral limb position sense, two point discrimination sense, tactile sense of positioning and obstacle graphics. If a patient is in the eyes closed cases, holding the hands of the objects, although they would feel, but can not determine the object's weight, size, shape, texture, and even the skin can not write simple number recognition, so , can not complete a comprehensive analysis of the object, called the loss of physical sense, it is back to the top of the central lobule after extensive destruction caused by the results. Cortical sensory dysfunction can be manifested as sensory neglect, when the stimulus affected limb, it feels completely normal or slightly decreased, while stimulating both limbs, such as, only the feelings caused by the contralateral limb, while the affected limb completely ignored. Ipsilateral limb when the tactile stimulation factors have been removed, the patient is still in a period of time feel the feeling stimulated by the continued existence of the phenomenon known as tactile retention. 2. Body image disturbance; patients autologous face difficulties in understanding the structure, which is especially prevalent right parietal lobe lesions, to elucidate the mechanism has not yet been fully understood. Many clinical manifestations, such as patients with paralysis of their own indifference, do not pay attention, seems to have nothing to do, there is no meaning of anxiety, called hemiplegia lost NOTE disease. The patient completely deny their hemiplegia, paralysis of the limbs even when the tips to the patients, strongly denied that it was his own body, sometimes considered to be someone else's hands or feet, or to explain the physical reasons unrelated to the reason is not in such phenomenon does not know the disease known as hemiplegia. Some patients had lost limbs in the sense that his body no longer exists, is not paralyzed his body, his limbs this has been lost. Another performance is more a physical feeling or more, this show is called phantom limb syndrome and more. Another patient had finger agnosia, disorientation around the body, such as autologous amnesia. 3. Loss of structural disorder; loss of structural disorder, also called structure apraxia. Refers to the structure of the arrangement of space objects, architecture, painting, design and other issues involving the relationship between space and can not correctly understand and identify, can not be combined, can not understand the relationship between each other and can not properly use the tools to work, resulting in life find it difficult to . Available clinical painting, building blocks and other methods to check the patient although sink, and each component may still, but the ratio between the lack of capacity and layout, or up and down upside down, overcrowded or decentralized arrangement, loss of original shape, no The concept of space, the lack of solid relationship. Resulting loss of structure, mechanism of disease has not yet achieved consensus. 4.Gerstmann syndrome; seen after the lower part of the angular gyrus parietal lobe, supramarginal gyrus in the occipital and parietal parts of transitional lesions, the clinical manifestations finger agnosia, loss of orientation about disease, agraphia, miscalculated the Lord. Finger agnosia most common, often bilateral, instruct patient to produce the specified fingers, the fingers can not be identified, using the chaos of the fingers, especially thumb, little finger, middle finger the most serious, finger agnosia Gerstmann syndrome is important components. About loss of directional disorder not only in the identification of self to others when they can not distinguish between left and right limbs, but about the surrounding environment, orientation is not necessarily affected. Agraphia, mainly it difficult to write, but can not read or copied obstacles. Acalculia barriers to written calculation significantly. 5. Alexia; the top of the left cerebral hemisphere occipital often caused by dyslexia, the reading disability, accompanied by writing disabilities. Dyslexia can be divided into two types: (1) subcortical alexia: the patient can not read written or printed text and can not be pronounced, but the spontaneous writing and copying from obstacles. Patients can rely on writing to express their thinking, but you can not read their own written articles or letters. Subcortical alexia often accompanied by hemianopia. (2) cortical alexia: Patients do not know and can not be read than text, but also often accompanied by written any errors, and not dictation, transcription and spontaneous writing. 6. Seizures; parietal lobe tumor, mostly due to the limitations of seizure onset, and often feel, performance for the onset of contralateral lesions paresthesia, starting with the thumb and index finger more common parts, but the feet were also starting not uncommon to paroxysmal numbness, electric shock-like sensation or pain-based, extended to the fixed direction, but was limited for the sport or clonic spasm, or sensory symptoms before the symptoms start following the onset of exercise, even evolve into grand mal epilepsy. Often point discrimination after the onset of sleep. Physical sensation, position sense and other transient sensory disturbance. 7. Hemiplegia or single paralysis; parietal lobe tumor lesions often occur contralateral limb paralysis, hemiplegia or single. Symptoms of paralysis is not the parietal lobe itself is invaded and the motor areas of the tumor due to forward. And paralysis symptoms the same time, we can see deep hyperreflexia, increased muscle tone but not obvious. 8. Other; parietal lobe tumor, sometimes there can be contralateral limb muscle atrophy lesions, visual distortion produced by optical illusion, the opposite 1 / 4 blind or with bits of hemianopia, and awareness of the geographical barriers between and so on. Secondary parietal skull plain film other parts of the tumor and the tumor cerebral hemisphere as, according to the different nature of the tumor, some tumors may also cause varying degrees of changes in the skull, such as meningiomas can cause within a small area of skull bone growth or damage to plate and so on. Parietal lobe tumors ultrasound ultrasound waves to the contralateral common with midline shift. EEG EEG parietal tumors performance characteristics are: the limitations of δ or θ wave wave appears broader, generally the top side of the main disease, spread to the occipital, temporal, the temporal ago, the amount of after inferior parietal and other parts, especially at the top of the brain falx tumors, slow the emergence of a wider, often difficult to distinguish tumor occipital and temporal. δ wave confined to the top or pillow top, there are likely to parietal lobe tumor. Deep parietal lobe tumor, usually not obvious limitations of δ wave, multi-display 5 to 7 times / second limitation θ scattered or continuous wave and lazy wave. Parietal lobe tumors easily lead to disease hemisphere (30%) or both sides of the hemisphere (about 10%) of the broad diffuse slow wave, sometimes generalized spike and wave display, and the case or the background on both sides of α wave often disorders. Cerebral angiography Cerebral angiography parietal lobe tumor characteristics: before and after the bit in the anterior cerebral artery as the shift was parallel to the side of the middle cerebral artery branch convex or partially separated from the down. In the artery as the lateral under pressure, where the parts of the tumor blood vessels under pressure. (1) parietal falx brain tumors: before and after the bit like the shift to the opposite side shows artery for the straight line, the remote shift significantly. Lateral side as the tumor before the show, the corpus of peripheral artery and its branches curved; tumor after partial arterial end of the week when the corpus has been under pressure to move or corpus of peripheral arterial pushed forward bending, straightening, or were terminal branches arc shape. Artery group of pushing forward a little down, or peripheral dispersion phenomenon. (2) parietal parasagittal tumor: as shown before and after the bit shift to the contralateral artery, the top was, under the pressure of the corpus of peripheral arterial branches curved, falx sign positive. Middle cerebral artery groups were under pressure to low. Lateral groups like the see the downward shift in the arteries, the corpus of peripheral arterial slightly down or flat, so place mostly meningiomas, Gu Chang said in the anterior cerebral artery and tortuous artery thickening. (3) frontoparietal tumors: before and after the show bit like an arc-shaped anterior cerebral artery was shifted to the opposite side. Middle cerebral artery group shifted slightly down. No changes in the blood vessels "○" shaped performance. Lateral middle cerebral artery, as shown flattened obvious pressure, changes in the amount of lifting arteries was significantly higher than other parts of the tumor, manifested by the extrusion, straightening, branch separated from each other and so on. Lateral ventricle due to the body making the rear triangle and the parietal lobe and the corresponding, it shows lateral parietal lobe tumors under the pressure of the body, septum pellucidum, third ventricle was shifted to the opposite side slash. After partial tumor close to the occipital, parietooccipital tumors that can be caused by both Delta and occipital horn down. CT brain CT examination of intracranial tumors, especially in the diagnosis of supratentorial tumors than other diagnosis techniques have more advantages, is the ideal method of diagnosis. Identification of (a) of chronic subdural hematoma; parietal chronic subdural hematoma is a common disease in both young common. The clinical course and brain tumors are similar, but have history of head injury, the more minor injuries, it is often forgotten by the patient and not take the initiative to tell, even when asked not remember history. After more than a few months or even years before clinical symptoms appear gradually. Early symptoms are usually headache, dizziness, epilepsy can also find out the limitations of sensory or sensory disability side of the body, careful examination can find some signs and symptoms of parietal lobe damage. Skull plain film examination may show chronic increased intracranial pressure, usually no fracture line. Ultrasound examination revealed midline wave shift (no shift may be bilateral). EEG slow wave appears to have limitations. Carotid angiography showed no vascular area can often confirm the diagnosis. (B) brain abscess; parietal lobe brain abscess compared with the incidence of temporal, frontal, far more than the blood-borne infection, acute expression of more than a systemic (fever, peripheral blood polymorphonuclear leukocytes as an increase) and acute intracranial pressure by other, lumbar puncture showed purulent CSF has changed, it is easy and differential diagnosis of brain tumors. However, chronic parietal lobe brain abscess, longer course, the similar clinical symptoms and parietal lobe tumors, most difficult to identify, only when surgical exploration to confirm th
e diagnosis. (C) cerebrovascular disease; artery lesions in the brain, such as vascular malformations, arteriovenous fistula, aneurysm, arteriosclerosis, etc., there may be parietal lobe syndrome. The top of the middle cerebral artery, temporal, occipital lobe occupies prone to ischemic softening. Dominant hemisphere angular gyrus softening beneath writing, reading and loss of Gerstmann syndrome occurs, care should be carefully examined and tumors. Prevention of prevention: primary prevention refers to health promotion and risk reduction factors. The first line of defense of the role is to promote the general population of healthy lifestyles, reducing exposure to harmful factors in the environment as a way to completely avoid the occurrence of cancer. According to current knowledge, in addition to prevention of air, water, food and workplace carcinogens and suspected carcinogens, the changing lifestyle of smoking, drinking and other bad habits are the primary prevention of the content.
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