17:23,10,Aug,2006 | (3486/0/0) | Original

omental infarction treatment

Idiopathic Omental segmental infarction in 1 case report
Omentum adhesions after appendectomy syndrome diagnosis and treatment of one case
1 case of pregnancy, the greater omentum
Pedicled greater omentum in the appendix Experience in the application root gangrene
Omental pregnancy care with hemorrhagic shock
Ultrasound diagnosis of omental metastases in 1 case report
Cases of omental 1 case of spontaneous hemorrhage
Omentum, mesenteric hemorrhage misdiagnosed as three cases of splenic rupture
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Intrahepatic cholangiocarcinoma after hepatic resection for intraoperative intravenous Omentum Efficacy of mitomycin C to prevent recurrence
Autologous transplantation of splenic omentum chip traumatic rupture of the spleen in resection of the use of (1)
Large subserosal uterine leiomyoma with vascular fusion in 1 case omentum
Great omentum in 1 case of metastasis
Congenital defect with omentum after the vault prolapse in 1 case
Application of greater omentum treatment of obstetric fistula in 1 case report
Omental adhesions with surrounding tissues, incarcerated hernia misdiagnosed as testicular torsion in 1 case
Pediatric Omental cyst torsion in 1 case report of ultrasonographic
Pediatric Omental cyst torsion in 1 case report of ultrasonographic
omental infarction treatment

The names of diseases: primary segmental omental infarction English name: Idiopathic Segmental Infarction; Spontaneous omental infarction; hemorrhagic omental infarction drug therapy: involvement of the omentum should be widely resected tissue. 【Overview】 basic primary segmental omental infarction (Idiopathic Segmental Infarction) is a rare occurrence of infarction and trauma, torsion, cardiovascular disease, adhesions or other abdominal pathology nothing to do, is unexplained acute retinal blood vessels disease, there is spontaneous omental infarction, or hemorrhagic infarction of the omentum said. 】 【Diagnosis because the patients abdominal pain, right lower quadrant tenderness, and muscle tension and other peritoneal irritation, and increased the number of peripheral blood white cell, often misdiagnosed as acute appendicitis, and even acute cholecystitis. B-mode ultrasound examination may detect a small amount of ascites, paracentesis may be out of bloody ascites. Often due to acute abdomen was diagnosed when laparotomy. Many patients the disease was misdiagnosed as acute appendicitis, but is found in abdominal surgery in tune with bloody serous effusion, had just noted, infarct region can form a hard, red or purple lumps. 】 【Treatment should be widely involved in the retinal tissue removed. 【ã€
Pathogenesis of many scholars with different theories to explain its pathogenesis, the general said: venous distention or abnormal venous drainage, heavy elongated retinal blood vessel and / or epithelial damage, mild trauma or abdominal pressure and other anatomical variations can cause retinal retinal thrombosis. Omental infarction is usually the right side of the free edge. Triangular mass lesions, the majority of a diameter of about 6 ~ 8cm, sometimes extend to the surrounding tissue and the parietal peritoneum. Were observed with retinal vein thrombosis and multinucleated cells and round cell infiltration. Common clinical manifestations】 【20 ~ 30-year-old well-nourished male, male-female 2.5 times, and obesity are closely related. Patients showed persistent severe right side abdominal pain, right lower abdominal pain about 3 / 4, activities have worsened, not often nausea and vomiting, may have fever. Check the limitations of abdominal tenderness, pain and muscle tension balcony, often in the right lower quadrant tenderness Maxwell Point and its environs, can reach full or partial abdominal mass. Skin hypersensitivity is specific to the signs of the disease. WBC normal or slightly increased. 】 【Differential diagnosis of acute appendicitis, acute cholecystitis phase identification.
(A) etiology
Is not clear, the incidence of this disease may be related to vascular lesions on the greater omentum, greater omentum can promote blood circulation disorders of the factors of acute venous disease than artery disease, clinical studies have shown that, in most cases, intravenous lesions are the main factors of this disease.
1. Omental vein disease mainly caused by the greater omentum for a variety of incentives to intimal injury, thrombosis, luminal occlusion of venous reflux disorder, thereby affecting the blood supply to the retina, these factors are:
(1) The sudden increase in intra-abdominal pressure: coughing, vomiting, defecation force, so that the veins within the retina by the sudden pull or swing.
(2) closed abdominal injury: even minor blunt injury, the greater omentum can also be subject to external forces, possible venous injury.
(3) Obesity: the greater omentum in obese patients have excess body fat deposition, its volume increases, weight gain, physical activity, the traction on the omentum and increase the intensity of movement.
(4) intravenous hypercoagulable state, may promote thrombosis.
(5) heart failure, affecting the retinal vein reflux.
2. Omental artery atherosclerotic lesions, nodular go far though mainly in the arteries within the artery, but may also spread to smaller omental artery, the occurrence of stenosis and occlusion of arteries, eventually leading to blood supply to areas of large retinal necrosis.
(B) of the
1. Omental lesion of idiopathic omental infarction mainly in the right part of the study found that clinical data and right side of the abnormal blood vessels over the left side of the greater omentum, and fat deposition, but also to the right side hypertrophy more.
2. Pathological features of omental infarction occurs often has a triangle, 2 ~ 20cm in diameter ranging from 6 ~ 8cm but were more common, local show edema, hemorrhage and necrosis, the appearance of dark red or dark purple, omental infarction often with extensive adhesions surrounding tissues and organs, mainly with the right colon, duodenum and pelvic wall adhesions, peritoneal cavity a small amount of bloody serous effusion, the course is longer, may be purulent exudate, microscope, large retinal vein thrombosis, inflammatory cell infiltration, omental torsion and external pressure without the appearance of the phenomenon can be other than the possibility of secondary necrosis of omentum.
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