07:59,15,Sep,2005 | (622/0/0) | Original

hypertrophic obstructive cardiomyopathy treatment


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hypertrophic obstructive cardiomyopathy treatment

Home> academic journals> Journal of Cardiovascular Research "-2004 2> Chemical ablation for the treatment of hypertrophic obstructive cardiomyopathy Status
Ablation in the treatment of hypertrophic obstructive cardiomyopathy Status
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Hypertrophic obstructive cardiomyopathy (Hyperyrophic ObstructiveCardiomyopthy, HOCM) is a kind of asymmetric septal hypertrophy, left ventricular outflow channel for the clinical features of primary obstructive cardiomyopathy. Clinically, many accompanied by exertional dyspnea, atypical pneumonia angina pectoris, syncope, sudden death and so on. Because the poor quality of life of patients, sudden death rate, how to actively and effectively improve the symptoms, prevention of sudden death has been a continuing concern that the treatment developed very rapidly. At present, the common treatment are: drug therapy (such as β-blockers, calcium antagonists), surgical treatment, right atrial-demand ventricular pacing (DDD pacing) therapy. transcatheter percutaneous use of ethanol septal coronary ablation (percutaneous transluminal septal myocardial abla-tion, PTSMA) treatment of hypertrophic obstructive cardiomyopathy intervention in recent years a new development, this paper this new approach are reviewed. of: Lu Jiang Jianjun of the first unit of the : 317000, Zhejiang Province, Linhai, Taizhou Hospital of Zhejiang Province, title: Chinese Journal of Cardiovascular English title: CHINESE JOURNAL OF CARDIOVASCULAR REVIEW, the volume (of): 2004 2 (2) Key words: R542.2 Key words: cardiac disease intervention ablation machine marked DOI: R54 R65 machine standard Key words: chemical ablation for the treatment of hypertrophic obstructive cardiomyopathy treatment of primary atypical angina, sudden death ventricular cardiomyopathy β blockers agent after surgical treatment of coronary artery ablation therapy clinical characteristics of ethanol-demand pacing intervention calcium antagonists improve the symptoms of dyspnea left ventricular Fund Project: DOI: References (16) Waller BF.Maron B J. Epstein SETransmural Myocardial infarction in hypertrophic cardiomypathy: a cause of conversion from left ventricular asymmetry to symmetry and from normal-sized to dilated left ventricular cavity 1981 Sigwart UNon-surgical myocardial reduction for hypertrophic obstructive cardio-myophathy 1995 Kuhn H. Gietzen F. Leuner CIndution of subaortic septal ischemia to reduce obstruction in hpertrophic obstructive cardiomyopathy: Study to develop a new catheter-based concept of treatment 1997 Faber L. Seggewiss H. Fassbender DCatheter treatment in hypertrophic obstruc-rive cardiomyopathy: identification of the perfusion area of septal branches by myocar -dial contrast echocardiography (MCE): first experiences 1997 (zk) Faber L. Seggewiss H. Gleichmann UPercutaneous transluminal septal myocardial ablation in hypertrophic obstructive crdiomyopathy: results with respect to intra-procedural myocardial contrast echocardiography 1998 Kaul SAssessment of coronary microcirculation with myocardial contrast echo-cardiography: current and future clinical application 1995 Liu Ying. LI accounting. Zhang coronary myocardial contrast echocardiography guidance of selective percutaneous septal myocardial ablation [Papers] - Journal of Ultrasound Medicine 2001 (10) Knight C. Kurbaan AS . Seggewis HNon-surgical septal reduction for hyper-trophic obstructive cardiomyopathy: Outcome in the first series of patients 1997 Gietzen F. Kuhn U. Leuner CAcute and longterm results after transcoronary ablation of septum hypertrophy in hypertrophic obstructive cardiomyopathy 1997 (zk) Zhaolin Yang. Wang Dawei . Yangpyeong chemical ablation catheter treatment of hypertrophic obstructive cardiomyopathy [Papers] - Cardiology 1998 (01) Seggewiss H. Faber L. Gleichmann UPercutaneous transluminal septal myocardial ablation in hypertrophic obstructive crdiomyopathy 1999 ablation of Liaoning Province technical coordination group. Lee accounted for percutaneous coronary septal ablation for hypertrophic obstructive cardiomyopathy term efficacy [Papers] - Cardiology 2001 (01) Faber L. Meissner A. Ziemssen PPercutaneous transluminal septal myocardial ablation for hypertrophic obstructive cardiomyopathy: long term follow up of the first series of 25 patients 2000 Yaling Han. Shou force. Wang Zulu hypertrophic obstructive cardiomyopathy ablation therapy (report of 8 cases) [Papers] - People's Liberation Army Medicine 2001 (12 ) Liu Li. Kwan Yu Ming. Liu Ying transcatheter septal branch during the first bolus Levovist induced ventricular fibrillation in 1 case [Papers] - Journal of Clinical Cardiology 2001 (07) Yan Ming Chau. Zhaolin Yang. Wei Dayu hypertrophic obstructive cardiomyopathy ablation catheter complications and management of cardiac arrhythmias [Papers] - Cardiac Arrhythmias 1999 (02)
>> More . similar to the literature cited relevant literature Bowen (1) Qiu Xing standard. Parties only. Hui Chen. Ye Ying. Xuying Jia. Houxu Min. Yuan Fang. Shaofeng off. Shihong Yu. Li Ruogu. Ni percutaneous septal immature side myocardial ablation in hypertrophic obstructive cardiomyopathy immediate effect and follow-up results [Papers] - CARDIOVASCULAR 2009 (4)
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yz *** hypertrophic obstructive cardiomyopathy, arrhythmia, first radiofrequency ablation or chemical ablation
1 #, and his exchange
Illnesses:
Hypertrophic obstructive cardiomyopathy, arrhythmia
Disease description (main symptom onset):
Sometimes flustered. (Frequency: about 2 times / month)
Almost every day there is a brief dizziness
2009-4 had gout. (I do not know with the "uniforms to metoprolol" causal relationship)
I am 40 years old mother in sudden death due to heart disease.
Big Brother Big Sister I have no similar symptoms.
And effectiveness of treatment was: dressed in uniforms, "metoprolol tartrate tablets" Betaloc (sooner or later one (25g))
Want kind of help: Wuxi Second People's Hospital, said doctors need to treat arrhythmia ablation, ablation of hypertrophic obstructive cardiomyopathy. But he said there was no certainty of success.
I ask Dr Chow:
Your hospital for sure how many of these diseases?
Cost and how much?
Two operations can be done with it? Do first the right one?
Surgery is successful, a total of how long to work?
Testing, test results :2002-6-21 echocardiography report:
1. Left atrium, left ventricular normal and asymmetric wall thickening in septal wall thickening and the former main outflow tract obstruction was no systolic performance (pressure 11.3mmHg), resting Color kinesis (CK) examination: no abnormal contraction, no abnormal mitral valve morphology activities, flow imaging and Doppler examination: mild reflux.
2. Aorta is not enlarged, thickened aortic valve does not open and close no abnormal color Doppler examination: no reflux.
3. Right atrium and right ventricle does not increase, the main pulmonary artery is not enlarged, color Doppler examination: no reflux.
4. Acoustic quantification (AQ) test ejection fraction 63%.
Ultrasonic Tip: hypertrophic cardiomyopathy (non-obstructive)
2008-7-11 echocardiography report:
1.3, range of motion reduced range of motion compensated left ventricular posterior wall enhancement, color Doppler: systolic left ventricular outflow tract velocity can see colorful shot, CW measured resting systolic blood flow velocity of left ventricular outflow tract 270CM / S, pressure 29mmHg, after exercise, 380CM / s, pressure 58mmHg.
2. Not thickened mitral valve, showing systolic forward movement, tissue Doppler display: diastolic mitral annulus peak of the spectrum E peak is greater than A, CDFI: Trace regurgitation the left atrium visible beam.
3. Tricuspid and pulmonary valve is not thickening, valve opening was no exception, CDFI: closed no reflux, pulmonary artery normal.
4. Aortic valve morphology and no abnormal valve opening and closing, the normal aorta, the main wave pump rate is still good, dicrotic wave exists.
5. Pericardial cavity no liquid dark area.
Ultrasonic Tip: hypertrophic obstructive cardiomyopathy
2010-3-30 echocardiography report:
Aortic root diameter of 28 (20 ~ 37)
Left ventricular end diastolic diameter 42 (35 ~ 56)
Left ventricular end systolic diameter of 27
38 Left atrial diameter (19 ~ 40)
Interventricular septum thickness of 25 ~ 26 (6 to 11)
Left ventricular posterior wall thickness 10 (6 to 11)
1. Left atrium, left ventricular normal and asymmetric left ventricular wall thickening was significantly thicker interventricular septum and anterior wall thickness of about 25 ~ 26mm, wall thickness of about 12 ~ 13mm, left ventricular outflow tract diameter of about 14mm, Color Doppler
This high-speed measurement and systolic turbulence, continuous Doppler and the maximum instantaneous systolic pressure of about 47mmHg.M type seen prior to the exercise systolic mitral valve (SAM sign). Resting color kinesis (CK) examination: no segmental contraction abnormalities, mitral valve leaflets, valve ring is not thickened, echo is not enhanced form of papillary muscles and tendons no abnormal activity, flap leaves open without restriction, closed well, color flow imaging and pulsed (continuous) Doppler examination: mild mitral regurgitation.
2. Not widened aortic root, aortic prosthetic leaves, no thickening of the valve ring, echo is not enhanced, open without restriction, closed well, color flow imaging and pulsed (continuous) Doppler: Aortic no reflux.
3. Right atrium and right ventricle does not increase pulmonary artery is not widened, continuous Doppler tricuspid regurgitation estimated systolic pulmonary artery pressure, according to 42mmHg.
4. Acoustic quantification (AQ) detection of left ventricular ejection fraction (EF) = 68%.
5. Left ventricular diastolic function parameters: mitral E peak velocity / tissue Doppler annular E 'peak velocity (E / E') = 8.0, E / A = 1.2
Ultrasonic tips: 1. Hypertrophic obstructive cardiomyopathy
2. Mildly elevated pulmonary artery pressure
2010-3-19 ECG:
Rhythm: sinus. Heart rate: 71 times. QRS mean electrical axis (can not read) PR interval: 10 seconds QRS duration: 10 seconds QT time: 37 seconds
Rr: sinus rhythm
Pre-hit syndrome.
2010-3-30 ECG:
Heart rate: 61bpm.
P wave duration: 86ms.
P-R interval: 130ms.
QRS time: 105ms.
QT / QTc: 424/420ms.
P-R-T: 0/-51/-117.
Diagnosis:
1. Is not complete right bundle branch block
2. Left ventricular hypertrophy
3.ST segment (down)-T wave changes
Visiting the hospital the last time: Wuxi People's Hospital
Posted at 2010-03-24 11:23:28 yz *** 2 # and his exchange
Added:
I am born in 1973.
Yesterday (2010-3-30) just went to the Wuxi People's Hospital check:
2010-3-30 echocardiography report:
Aortic root diameter of 28 (20 ~ 37)
Left ventricular end diastolic diameter 42 (35 ~ 56)
Left ventricular end systolic diameter of 27
38 Left atrial diameter (19 ~ 40)
Interventricular septum thickness of 25 ~ 26 (6 to 11)
Left ventricular posterior wall thickness 10 (6 to 11)
1. Left atrium, left ventricular normal and asymmetric left ventricular wall thickening was significantly thicker interventricular septum and anterior wall thickness of about 25 ~ 26mm, wall thickness of about 12 ~ 13mm, left ventricular outflow tract diameter of about 14mm, Color Doppler
This high-speed measurement and systolic turbulence, continuous Doppler and the maximum instantaneous systolic pressure of about 47mmHg.M type seen prior to the exercise systolic mitral valve (SAM sign). Resting color kinesis (CK) examination: no segmental contraction abnormalities, mitral valve leaflets, valve ring is not thickened, echo is not enhanced form of papillary muscles and tendons no abnormal activity, flap leaves open without restriction, closed well, color flow imaging and pulsed (continuous) Doppler examination: mild mitral regurgitation.
2. Not widened aortic root, aortic prosthetic leaves, no thickening of the valve ring, echo is not enhanced, open without restriction, closed well, color flow imaging and pulsed (continuous) Doppler: Aortic no reflux.
3. Right atrium and right ventricle does not increase pulmonary artery is not widened, continuous Doppler tricuspid regurgitation estimated systolic pulmonary artery pressure, according to 42mmHg.
4. Acoustic quantification (AQ) detection of left ventricular ejection fraction (EF) = 68%.
5. Left ventricular diastolic function parameters: mitral E peak velocity / tissue Doppler annular E 'peak velocity (E / E') = 8.0, E / A = 1.2
Ultrasonic tips: 1. Hypertrophic obstructive cardiomyopathy
2. Mildly elevated pulmonary artery pressure
2010-3-30 ECG:
Heart rate: 61bpm.
P wave duration: 86ms.
P-R interval: 130ms.
QRS time: 105ms.
QT / QTc: 424/420ms.
P-R-T: 0/-51/-117.
Diagnosis:
1. Is not complete right bundle branch block
2. Left ventricular hypertrophy
3.ST segment (down)-T wave changes
Posted at 2010-03-31 11:27:05 Doctor weeks of the new Re: Hypertrophic obstructive cardiomyopathy, irregular heart beats, the first radiofrequency ablation or chemical ablation
# 3 of Cardiology, Shanghai Zhongshan Hospital Chief Physician
Your age is 7 years old? Zhou, Zhongshan Hospital of Shanghai reach new
Weeks of the new doctor
I published in 2010-04-03 18:30:35 yz *** 4 #, and his exchange
Born in 1973.
Posted at 2010-04-04 08:05:18 #, weeks up to a new doctor 5 Department of Cardiology, Zhongshan Hospital, Shanghai chief physician
Depending on your situation, you can consider choosing invasive therapy, such as ablation, pacemaker, surgical treatment, but the effect is personal, Shanghai Zhongshan Hospital of weeks of the new
Weeks of the new doctor I published in 2010-04-04 20:14:13 yz *** 6 #, and his exchange
Thank you. Surgery is too dangerous, the costs are high.
Posted at 2010-04-04 20:55:24 yz *** 7 #, and his exchange
Dr Chow, chemical ablation, the ECG suggest a "sinus rhythm, pre-strike syndrome" can also be cured?
Posted at 2010-04-14 13:58:28 #, weeks up to a new doctor 8 Department of Cardiology, Zhongshan Hospital, Shanghai chief physician
Yes, but a clear diagnosis. Zhou, Zhongshan Hospital of Shanghai reach new
Weeks of the new doctor I Posted 2010-04-21 21:45:23
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